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Complex I activity may be selectively altered in later stages of BD when increased oxidative stress is more prominent, as elevated oxidative stress leads to ETC dysfunction, generating more oxidative stress and creating a vicious cycle [31]

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Several options are available for pharmacological inhibition of glucose metabolism: glucose uptake (phloretin) (150) and glycolytic enzymes (3-bromopyruvate and Koningic acid for GAPDH) (151, 152)

A list of DNA primers used in this study is presented in Supplementary Table 1

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